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Gene polymorphisms among the key elements of the coagulation and fibrinolytic cascade appear to influence the effects of estrogen on risk for venous thromboembolic events and possibly arterial thrombosis as well. Emerging data also suggest that allelic variants in the estrogen receptor may modulate estrogen’s effects, especially with respect to bone and lipid metabolism. In conclusion, within isolated mitochondria from preconditioned hearts, is increased and in concert with enhanced.
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